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A Possible Mechanism for the Anti-Proliferation of Gold Drugs on Endothelial Cells Involving Inhibition of Proto-Oncogeneproducts

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Angiogenesis in Health and Disease

Part of the book series: NATO ASI Series ((NSSA,volume 227))

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Abstract

The unusual persistence of the inflammatory process in rheumatoid arthritis has repeatedly been correlated with the continuous growth and proliferation of microvascular endothelial cells (EC), constituting synovial angiogenesis.

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© 1992 Springer Science+Business Media New York

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Sahinoglu, T., Roncalli, M., Springall, D., Polak, J.M., Blake, D.R. (1992). A Possible Mechanism for the Anti-Proliferation of Gold Drugs on Endothelial Cells Involving Inhibition of Proto-Oncogeneproducts. In: Maragoudakis, M.E., Gullino, P., Lelkes, P.I. (eds) Angiogenesis in Health and Disease. NATO ASI Series, vol 227. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-3358-0_43

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  • DOI: https://doi.org/10.1007/978-1-4615-3358-0_43

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4613-6481-8

  • Online ISBN: 978-1-4615-3358-0

  • eBook Packages: Springer Book Archive

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