Abstract
The metabolic response during acute pancreatitis may depend on the one hand on acute stress, which is similar to that seen in other diseases such as sepsis or clinical situations such as surgical interventions, and on the other hand on damage to the Langerhans’s islets related to pancreatic inflammation. Hyperglycemia develops rather often in the early phase of acute pancreatitis, mainly in patients with severe disease [1–3]. This hyperglycemia could thus be the result of a hyperglucagonemia secondary to stress or the result of decreased synthesis and release of insulin secondary to the damage of pancreatic β-cells [4–7].
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© 1997 Springer-Verlag Berlin Heidelberg
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Domínguez-Muñoz, J.E., di Sebastiano, P., Malfertheiner, P. (1997). Endocrine Pancreatic Function During and Following Acute Pancreatitis. In: Malfertheiner, P., Domínguez-Muñoz, J.E., Schulz, HU., Lippert, H. (eds) Diagnostic Procedures in Pancreatic Disease. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-60580-2_11
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DOI: https://doi.org/10.1007/978-3-642-60580-2_11
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