Abstract
Among the abnormal fibrinogens which we recently analyzed, two types of abnormality were found to have two structural defects: a genetically determined amino acid substitution and an additional secondary modification of the molecule due to the mutation (1, 2). Here we briefly describe these abnormal molecules.
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Matsuda, M., Terukina, S., Yoshida, N., Yamazumi, K., Maekawa, H. (1990). Abnormal Fibrinogens with Two Structural Defects. In: Liu, C.Y., Chien, S. (eds) Fibrinogen, Thrombosis, Coagulation, and Fibrinolysis. Advances in Experimental Medicine and Biology, vol 281. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-3806-6_6
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DOI: https://doi.org/10.1007/978-1-4615-3806-6_6
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