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The Possibility of a Specific Calcium Sensor/Receptor Function of Proximal Renal Tubular Cells

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Urolithiasis

Abstract

Disturbances of calcium metabolism are well documented in renal calcium-stone disease and hypercalciuria is the most commonly-occurring metabolic deviation (1). Idiopathic hypercalciuria is considered to be caused by an increased gastrointestinal absorption of calcium, whereas a primary renal leak of calcium with the development of secondary hyperparathyroidism (HPT) is unusual, as judged by present diagnostic tools.

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References

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Author information

Authors and Affiliations

  1. Departments of Internal Medicine, Surgery, and Medical and Physiological Chemistry, University Hospital, Uppsala, Sweden

    B. Fellström, C. Juhlin, H. Johansson, L. Klareskog, S. Ljunghall, J. Rastad & G. Akerström

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  1. B. Fellström
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  2. C. Juhlin
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  3. H. Johansson
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  4. L. Klareskog
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  5. S. Ljunghall
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  6. J. Rastad
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  7. G. Akerström
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Editor information

Editors and Affiliations

  1. University of British Columbia, Vancouver, British Columbia, Canada

    Valerie R. Walker , Roger A. L. Sutton  & E. C. Bert Cameron ,  & 

  2. Southwestern Medical Center, University of Texas, Dallas, Texas, USA

    Charles Y. C. Pak

  3. King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia

    William G. Robertson

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© 1989 Springer Science+Business Media New York

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Fellström, B. et al. (1989). The Possibility of a Specific Calcium Sensor/Receptor Function of Proximal Renal Tubular Cells. In: Walker, V.R., Sutton, R.A.L., Cameron, E.C.B., Pak, C.Y.C., Robertson, W.G. (eds) Urolithiasis. Springer, Boston, MA. https://doi.org/10.1007/978-1-4899-0873-5_3

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  • DOI: https://doi.org/10.1007/978-1-4899-0873-5_3

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4899-0875-9

  • Online ISBN: 978-1-4899-0873-5

  • eBook Packages: Springer Book Archive

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