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PET in the Evaluation of Myocardial Infarction

Observations in Acute and Chronic Stage

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Clinical efficacy of positron emission tomography

Part of the book series: Developments in Nuclear Medicine ((DNUM,volume 12))

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Abstract

Coronary occlusion causes a sudden cessation of blood flow in the areas distal to the occlusion site starting a dynamic process of bio-electrical, biochemical, (ultra)structural and contractile changes. During this evolutionary process, progressing in a “wave-front”-1ike manner (Reimer et al., 1977) from endo-to epicardium initially reversible phases of ischemic injury lead to the final stage of completed infarction so that cellular viability is irreversibly lost.

Operated for the US Department of Energy by the University of California under contract no. DE-AC03-76-SF00012. This work was supported in part by the Director of the Office of Energy Research, Office of Health and Environmental Research, by NIH grant nos. HL 29845 and HL 33177, and by an Investigatorship Group Award by the Greater Los Angeles Affiliate of the American Heart Association.

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© 1987 ECSC, EEC, EAEC, Brussels-Luxembourg

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Sochor, H., Schwaiger, M., Brunken, R. (1987). PET in the Evaluation of Myocardial Infarction. In: Heiss, WD., Pawlik, G., Herholz, K., Wienhard, K. (eds) Clinical efficacy of positron emission tomography. Developments in Nuclear Medicine, vol 12. Springer, Dordrecht. https://doi.org/10.1007/978-94-009-3345-3_29

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  • DOI: https://doi.org/10.1007/978-94-009-3345-3_29

  • Publisher Name: Springer, Dordrecht

  • Print ISBN: 978-94-010-8002-6

  • Online ISBN: 978-94-009-3345-3

  • eBook Packages: Springer Book Archive

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